Codeine 15 mg


An important objective for improving patients’ quality of life and their capacity to participate in daily activities is the reduction of pain (analgesia). Codeine is an opioid analgesic that was first authorized in the United States in 1950. It reduces pain by raising the pain threshold without affecting consciousness or additional sense capabilities. Opiates like codeine are made from the Papaver somniferum plant, which is a type of poppy.

Codeine 15 mg is prescribed for some conditions that cause persistent coughing and is used as a peripheral pain reliever, hypnotic, sedating, antinociceptive, and antiperistaltic drug.

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One usual dosage type of this medication is codeine sulfate. When using an opioid-based painkiller is suitable, it is obtainable in tablet form and is intended for the alleviation of mild to fairly severe pain.

The solution formula is used as a cough reliever in individuals over the age of 18, either by itself or in combination with other medications in a syrup.

Codeine, which is comparable to morphine and hydrocodone in strength, is a mild narcotic painkiller and cough reliever. The body converts just a small portion of the consumed codeine to morphine. Codeine makes pain more tolerable, easing discomfort already present. Codeine not only lessens pain but also induces sedation, sleepiness, and reduced breathing.

Antitussive Properties

In studies, this medication had antitussive activity 6 and was successful in treating cough brought on by tuberculosis and cough-related sleeplessness. A direct impact of codeine on the center for coughs in the medulla causes it to block the cough response.

Intestinal Motility Effects

The motility of the bladder may be decreased by codeine via either a local or potentially centralized mechanism of action. Constipation could result from this.

Implications for the Central Nervous System

Despite being significantly less effective as a painkiller than morphine, codeine monohydrate is an opioid painkiller with similar applications. The mu opioid receptors in the body, located all across the central nervous system, are where it mostly acts. Codeine’s sedative properties aren’t as strong as morphine’s. By activating opioid receptors at particular places in the central nervous system, codeine could generate respiratory system depression.

Blood Pressure Effects

Because of peripheral blood vessel dilation and other processes, this medication carries a higher risk of compromising the capacity to regulate blood pressure.

Impacts on the pain caused by cancer and different kinds of pain

Codeine is an opioid analgesic with uses that are comparable to those of morphine.

Nonetheless, it has significantly less effective pain-relieving abilities. The mu receptors for opioids, which are found all over the central nervous system, are where they mostly act. Usually, an activity lasts for four hours.

It has been demonstrated repeatedly that opioid analgesics, such as codeine, ameliorate symptoms in cancer patients with severe discomfort.

A Method of Action

Although codeine is an agonist that is specifically designed for the mu opioid receptor, it has far lower selectivity than the more powerful opioid medication morphine. Codeine attaches to the mu opioid receptors, which let pain spread across the body via the central nervous system. Codeine’s conversion to morphine is supposed to give it its analgesic qualities.


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